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Ophthalmology

Lesions of the Visual Pathway

Built from Khurana — Neuro-Ophthalmology

The first 25 slides of Lesions of the Visual Pathway
The first 25 slides, exactly as they appear. The full deck has 63 content slides.

What’s inside

6 sections · 63 slides

  1. 01

    Overview

    • Why the visual pathway is a map you can read
    • Roadmap of this deck
    • Levels of the visual pathway and their field defects

    3 slides

  2. 02

    Optic Nerve Lesions

    Damage before the crossing — one eye is affected

    • Optic nerve lesions: the basics
    • Distal vs proximal optic nerve lesion

    2 slides

  3. 03

    Chiasmal Syndrome

    The crossing point — where nasal fibres decussate

    • What the chiasma does and why lesions here matter
    • Three types of chiasmal syndrome
    • Chiasmal fibre crossing and junctional scotoma
    • Field pattern from a chiasmal lesion
    • Chiasmal syndrome: etiology
    • Chiasmal syndrome: symptoms the patient reports
    • Hemifield slide and postfixation blindness
    • Chiasmal syndrome: the signs you find
    • Optic disc and field changes in chiasmal disease
    • Field defects in chiasmal disorders — the menu
    • Junctional scotoma pattern
    • Bitemporal hemianopia — the signature chiasmal defect
    • Common causes of central chiasmal lesions (approx. %)
    • Homonymous and binasal defects from the chiasmal region
    • Incongruous homonymous hemianopia
    • Bitemporal versus binasal field loss
    • Endocrine clues to a pituitary chiasmal lesion
    • Diagnosing and treating chiasmal syndrome
    • Neuroimaging of a sellar mass
    • The ophthalmologist's role after treatment
    • Visual field recovery after pituitary surgery
    • OCT retinal nerve fibre layer for prognosis

    22 slides

  4. 04

    Retrochiasmal Lesions

    Behind the crossing — homonymous defects that grow more congruous

    • The unifying rule behind the chiasma
    • Congruous versus incongruous homonymous defects
    • Tumour vs vascular: how homonymous defects behave
    • Optic tract syndrome — two flavours
    • Optic tract syndrome: Type I vs Type II
    • Bow-tie (band) optic atrophy
    • Field defect of optic tract syndrome
    • Lateral geniculate nucleus (LGN) lesions
    • Sectoranopia from geniculate infarction
    • Optic radiations: temporal lobe lesions
    • Meyer's loop and the pie-in-the-sky defect
    • Systemic features of temporal lobe lesions
    • Homonymous hemianopia denser superiorly
    • Optic radiations: parietal lobe lesions
    • Pie-in-the-floor inferior quadrantanopia
    • Neurologic features of parietal lobe lesions
    • Homonymous hemianopia denser inferiorly

    17 slides

  5. 05

    Lesions of the Visual Cortex

    The occipital lobe — the most congruous defects of all

    • How occipital lesions announce themselves
    • Central homonymous hemianopia
    • Temporal crescent — the occipital lobe's unique defect
    • Temporal crescent representation in the cortex
    • Macular-sparing homonymous hemianopia
    • Bilateral occipital lobe lesions
    • Ring scotoma and checkerboard defects
    • Other manifestations of occipital lobe lesions

    8 slides

  6. 06

    Putting It Together

    Reading the field defect back to the lesion

    • Localising by field defect — one-glance summary
    • Key takeaways
    • Why does a central chiasmal lesion cause bitemporal hemianopia?
    • References
    • Neuro-Ophthalmology

    5 slides