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Dermatology

UlcersAriela Hafner and Eli Sprecher

Built from Dermatology, 5th Edition

What’s inside

9 sections · 156 slides

  1. 01

    Overview

    • What this topic covers
    • Terms you will meet
    • Key features
    • $3B/yr

    4 slides

  2. 02

    Classifying Leg Ulcers

    A wound reaching through the epidermis and dermis, and the three-way split used to find its cause

    • What makes a wound chronic
    • Classification of causes of leg ulcers
    • Three main types of leg ulcer
    • Telling the three types apart
    • Comparing venous, arterial, and neuropathic leg ulcers — Venous
    • Comparing venous, arterial, and neuropathic leg ulcers — Arterial
    • Comparing venous, arterial, and neuropathic leg ulcers — Neuropathic/mal perforans

    7 slides

  3. 03

    Venous Ulcers

    The most common leg ulcer, caused by sustained high pressure in the vein system

    • How common is chronic venous disease
    • Normal blood flow up the leg
    • Normal versus incompetent venous valve flow
    • Venous hypertension: the shared endpoint
    • From venous hypertension to skin damage
    • Older theories of ulcer formation
    • Theories of venous ulcer formation
    • Why hemosiderin turns skin brown
    • Symptoms of venous disease
    • Where and how venous ulcers look
    • Venous ulcer with stasis dermatitis
    • Venous ulcer of the medial malleolus
    • Four other skin clues of venous hypertension
    • Cutaneous findings of venous hypertension
    • Hemosiderin discoloration of the shins
    • Atrophie blanche
    • Lipodermatosclerosis
    • Lipodermatosclerosis of the pannus
    • Acroangiodermatitis (pseudo-Kaposi sarcoma)
    • Grading venous disease: the CEAP system
    • What a biopsy shows
    • Ruling out other causes
    • History and physical examination of a leg ulcer
    • Livedoid vasculopathy and clotting risk
    • Livedoid vasculopathy
    • Why check for a clotting disorder
    • Thrombophilia and hypercoagulability evaluation
    • General principles of ulcer care
    • Moisture beats dryness
    • Debridement: clearing dead tissue
    • Sharp and mechanical debridement
    • Autolytic and enzymatic debridement
    • Maggot debridement therapy
    • Choosing a wound dressing
    • Types of wound dressings — Properties
    • Types of wound dressings — Properties (continued)
    • Types of wound dressings — Disadvantages
    • Types of wound dressings — Indications
    • Colonization versus true infection
    • Recognizing a truly infected wound
    • Topical antimicrobials
    • Silver as an antiseptic
    • Don't forget tetanus
    • Compression: the backbone of venous ulcer care
    • Classes of compression stockings
    • Types of compression therapy — Advantages
    • Types of compression therapy — Disadvantages
    • Classes of compression bandages
    • Classes of compression bandages (continued)
    • When compression can harm
    • Manual lymph drainage
    • Negative pressure wound therapy (VAC)
    • Skin substitutes
    • Topical and oral medicines
    • Approach to the venous ulcer patient

    55 slides

  4. 04

    Lymphedema

    Fluid buildup from poor lymph drainage, and the skin changes that follow

    • Lymphedema versus ordinary edema
    • Primary lymphedema by age of onset
    • Bilateral primary lymphedema (Milroy disease)
    • Genetic causes of primary lymphedema
    • Causes of lymphedema
    • Causes of lymphedema (continued)
    • Secondary lymphedema causes
    • Secondary lymphedema from obesity and myxedema
    • The lymphedema-infection cycle
    • How lymphedema progresses
    • Elephantiasis nostras verrucosa
    • Elephantiasis nostras verrucosa
    • Fibrotic papules in elephantiasis nostras verrucosa
    • Treating lymphedema and its complications
    • Other conditions that mimic lymphedema
    • Lipedema of the lower legs

    16 slides

  5. 05

    Arterial Ulcers

    Ulcers from poor artery blood flow: peripheral artery disease

    • Peripheral artery disease (PAD) basics
    • Why poor blood flow causes ulcers
    • Symptoms as PAD worsens
    • What an arterial ulcer looks like
    • Arterial ulcer
    • Other clues of poor artery flow
    • Measuring the ankle-brachial index (ABI)
    • Determination and interpretation of the ABI
    • Other tests for artery disease
    • Look-alikes of arterial ulcers
    • Cholesterol emboli
    • Treating arterial ulcers

    12 slides

  6. 06

    Diabetic and Neuropathic Ulcers

    Nerve damage, not just poor circulation, is the leading cause of diabetic foot ulcers

    • A major burden of diabetes
    • Three types of diabetic neuropathy
    • Consequences of diabetic neuropathy
    • PAD adds to diabetic ulcer risk
    • What a diabetic ulcer looks like
    • Neuropathic (mal perforans) ulcers
    • Mal perforans ulcers of the great toes
    • Working up a diabetic foot ulcer
    • Off-loading: taking pressure off the ulcer
    • Wound care specifics for diabetic ulcers
    • Growth factors and skin substitutes
    • Preventing diabetic foot ulcers

    12 slides

  7. 07

    Pressure Ulcers

    Ulcers from sustained pressure on skin trapped between a bone and an outer surface

    • What is a pressure ulcer
    • Common sites for pressure ulcers
    • How common and costly
    • Four causes of pressure ulcers
    • Pressure and time both matter
    • How shear and friction add to damage
    • Staging pressure ulcers: Stage I and II
    • Staging pressure ulcers: Stage III and IV
    • NPUAP pressure ulcer staging
    • Stage III sacral pressure ulcer
    • Black eschar of the heel
    • Sacral decubitus ulcer
    • Pressure ulcer in a wheelchair-bound patient
    • What the biopsy shows
    • Preventing and treating pressure ulcers
    • Treatment by stage

    16 slides

  8. 08

    Other Causes of Skin Ulceration

    Rarer causes to consider when an ulcer resists standard treatment

    • When to think 'rare cause'
    • Diffuse dermal angiomatosis
    • Blood disorders and ulcers
    • Phlegmasia cerulea dolens
    • Tropical ulcers
    • A wide range of additional causes
    • Additional causes of cutaneous ulceration
    • Diffuse dermal angiomatosis
    • Drug-related ulcers
    • A genetic cause: prolidase deficiency
    • Prolidase deficiency with spontaneous ulcers
    • Reference table lead-in
    • Additional causes of cutaneous ulceration
    • Additional causes of cutaneous ulceration (continued)
    • Additional causes of cutaneous ulceration (continued)
    • Additional causes of cutaneous ulceration (continued)
    • Additional causes of cutaneous ulceration (continued)
    • Additional causes of cutaneous ulceration (continued)
    • Additional causes of cutaneous ulceration (continued)
    • Additional causes of cutaneous ulceration (continued)

    20 slides

  9. 09

    Putting It Together

    The big ideas to carry forward from this chapter on ulcers

    • Match the exam to the cause
    • Two goals of treatment, every time
    • Prevention pays off
    • References
    • References (continued 2/10)
    • References (continued 3/10)
    • References (continued 4/10)
    • References (continued 5/10)
    • References (continued 6/10)
    • References (continued 7/10)
    • References (continued 8/10)
    • References (continued 9/10)
    • References (continued 10/10)
    • Dermatology, 5th Edition (2-Volume Set)

    14 slides