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Dermatology

UlcersAriela Hafner and Eli Sprecher

Built from Dermatology, 5th Edition

The first 25 slides of UlcersAriela Hafner and Eli Sprecher
The first 25 slides, exactly as they appear. The full deck has 156 content slides.

What’s inside

9 sections · 156 slides

  1. 01

    Overview

    • What this topic covers
    • Terms you will meet
    • Key features
    • $3B/yr

    4 slides

  2. 02

    Classifying Leg Ulcers

    A wound reaching through the epidermis and dermis, and the three-way split used to find its cause

    • What makes a wound chronic
    • Classification of causes of leg ulcers
    • Three main types of leg ulcer
    • Telling the three types apart
    • Comparing venous, arterial, and neuropathic leg ulcers — Venous
    • Comparing venous, arterial, and neuropathic leg ulcers — Arterial
    • Comparing venous, arterial, and neuropathic leg ulcers — Neuropathic/mal perforans

    7 slides

  3. 03

    Venous Ulcers

    The most common leg ulcer, caused by sustained high pressure in the vein system

    • How common is chronic venous disease
    • Normal blood flow up the leg
    • Normal versus incompetent venous valve flow
    • Venous hypertension: the shared endpoint
    • From venous hypertension to skin damage
    • Older theories of ulcer formation
    • Theories of venous ulcer formation
    • Why hemosiderin turns skin brown
    • Symptoms of venous disease
    • Where and how venous ulcers look
    • Venous ulcer with stasis dermatitis
    • Venous ulcer of the medial malleolus
    • Four other skin clues of venous hypertension
    • Cutaneous findings of venous hypertension
    • Hemosiderin discoloration of the shins
    • Atrophie blanche
    • Lipodermatosclerosis
    • Lipodermatosclerosis of the pannus
    • Acroangiodermatitis (pseudo-Kaposi sarcoma)
    • Grading venous disease: the CEAP system
    • What a biopsy shows
    • Ruling out other causes
    • History and physical examination of a leg ulcer
    • Livedoid vasculopathy and clotting risk
    • Livedoid vasculopathy
    • Why check for a clotting disorder
    • Thrombophilia and hypercoagulability evaluation
    • General principles of ulcer care
    • Moisture beats dryness
    • Debridement: clearing dead tissue
    • Sharp and mechanical debridement
    • Autolytic and enzymatic debridement
    • Maggot debridement therapy
    • Choosing a wound dressing
    • Types of wound dressings — Properties
    • Types of wound dressings — Properties (continued)
    • Types of wound dressings — Disadvantages
    • Types of wound dressings — Indications
    • Colonization versus true infection
    • Recognizing a truly infected wound
    • Topical antimicrobials
    • Silver as an antiseptic
    • Don't forget tetanus
    • Compression: the backbone of venous ulcer care
    • Classes of compression stockings
    • Types of compression therapy — Advantages
    • Types of compression therapy — Disadvantages
    • Classes of compression bandages
    • Classes of compression bandages (continued)
    • When compression can harm
    • Manual lymph drainage
    • Negative pressure wound therapy (VAC)
    • Skin substitutes
    • Topical and oral medicines
    • Approach to the venous ulcer patient

    55 slides

  4. 04

    Lymphedema

    Fluid buildup from poor lymph drainage, and the skin changes that follow

    • Lymphedema versus ordinary edema
    • Primary lymphedema by age of onset
    • Bilateral primary lymphedema (Milroy disease)
    • Genetic causes of primary lymphedema
    • Causes of lymphedema
    • Causes of lymphedema (continued)
    • Secondary lymphedema causes
    • Secondary lymphedema from obesity and myxedema
    • The lymphedema-infection cycle
    • How lymphedema progresses
    • Elephantiasis nostras verrucosa
    • Elephantiasis nostras verrucosa
    • Fibrotic papules in elephantiasis nostras verrucosa
    • Treating lymphedema and its complications
    • Other conditions that mimic lymphedema
    • Lipedema of the lower legs

    16 slides

  5. 05

    Arterial Ulcers

    Ulcers from poor artery blood flow: peripheral artery disease

    • Peripheral artery disease (PAD) basics
    • Why poor blood flow causes ulcers
    • Symptoms as PAD worsens
    • What an arterial ulcer looks like
    • Arterial ulcer
    • Other clues of poor artery flow
    • Measuring the ankle-brachial index (ABI)
    • Determination and interpretation of the ABI
    • Other tests for artery disease
    • Look-alikes of arterial ulcers
    • Cholesterol emboli
    • Treating arterial ulcers

    12 slides

  6. 06

    Diabetic and Neuropathic Ulcers

    Nerve damage, not just poor circulation, is the leading cause of diabetic foot ulcers

    • A major burden of diabetes
    • Three types of diabetic neuropathy
    • Consequences of diabetic neuropathy
    • PAD adds to diabetic ulcer risk
    • What a diabetic ulcer looks like
    • Neuropathic (mal perforans) ulcers
    • Mal perforans ulcers of the great toes
    • Working up a diabetic foot ulcer
    • Off-loading: taking pressure off the ulcer
    • Wound care specifics for diabetic ulcers
    • Growth factors and skin substitutes
    • Preventing diabetic foot ulcers

    12 slides

  7. 07

    Pressure Ulcers

    Ulcers from sustained pressure on skin trapped between a bone and an outer surface

    • What is a pressure ulcer
    • Common sites for pressure ulcers
    • How common and costly
    • Four causes of pressure ulcers
    • Pressure and time both matter
    • How shear and friction add to damage
    • Staging pressure ulcers: Stage I and II
    • Staging pressure ulcers: Stage III and IV
    • NPUAP pressure ulcer staging
    • Stage III sacral pressure ulcer
    • Black eschar of the heel
    • Sacral decubitus ulcer
    • Pressure ulcer in a wheelchair-bound patient
    • What the biopsy shows
    • Preventing and treating pressure ulcers
    • Treatment by stage

    16 slides

  8. 08

    Other Causes of Skin Ulceration

    Rarer causes to consider when an ulcer resists standard treatment

    • When to think 'rare cause'
    • Diffuse dermal angiomatosis
    • Blood disorders and ulcers
    • Phlegmasia cerulea dolens
    • Tropical ulcers
    • A wide range of additional causes
    • Additional causes of cutaneous ulceration
    • Diffuse dermal angiomatosis
    • Drug-related ulcers
    • A genetic cause: prolidase deficiency
    • Prolidase deficiency with spontaneous ulcers
    • Reference table lead-in
    • Additional causes of cutaneous ulceration
    • Additional causes of cutaneous ulceration (continued)
    • Additional causes of cutaneous ulceration (continued)
    • Additional causes of cutaneous ulceration (continued)
    • Additional causes of cutaneous ulceration (continued)
    • Additional causes of cutaneous ulceration (continued)
    • Additional causes of cutaneous ulceration (continued)
    • Additional causes of cutaneous ulceration (continued)

    20 slides

  9. 09

    Putting It Together

    The big ideas to carry forward from this chapter on ulcers

    • Match the exam to the cause
    • Two goals of treatment, every time
    • Prevention pays off
    • References
    • References (continued 2/10)
    • References (continued 3/10)
    • References (continued 4/10)
    • References (continued 5/10)
    • References (continued 6/10)
    • References (continued 7/10)
    • References (continued 8/10)
    • References (continued 9/10)
    • References (continued 10/10)
    • Dermatology, 5th Edition (2-Volume Set)

    14 slides