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The first 25 slides, exactly as they appear. The full deck has 47 content slides.
Ophthalmology
Traumatic Optic Neuropathy
Built from Khurana — Neuro-Ophthalmology
What’s inside
8 sections · 47 slides
Overview
- What you will learn in this deck
What traumatic optic neuropathy is
- The idea in plain terms
- The core split: direct versus indirect injury
- Who gets it, and from what
The anatomy that sets the trap
- The optic nerve, and the cells it carries
- Three wrappings, borrowed from the brain
- The four parts of the optic nerve
- Why the intraorbital nerve is deliberately slack
- The choke point: where the nerve is pinned to bone
- Two more places trauma can strike
Recognising and evaluating it
- How it presents
- A checklist for the suspected case
- Stabilise the patient first
- What the examination looks for
- Peripapillary haemorrhage after optic nerve injury
- Electrophysiology: reading the electrical signal
- ERG and neuroimaging
Why the nerve keeps dying
- Direct injury: uncommon, and why
- Indirect injury runs in two waves
- Primary mechanism: the first, physical blow
- What the mechanical forces actually do
- Secondary mechanism: the cascade that follows
- The biochemical machinery of the second wave
Treatment and its controversy
- Three options, none of them certain
- The rationale for steroids
- Where the steroid idea came from: NASCIS 2
- But does spinal-cord evidence transfer to the eye?
- The warning from the CRASH trial
- What the Cochrane review concluded
- Surgery: the compartment-syndrome idea
- Surgical approaches and sheath fenestration
- Weighing benefit against harm
Recent advances and current research
- New directions being explored
Pulling it together
- Traumatic optic neuropathy at a glance
- Clinical hooks worth remembering
- No perception of light after frontal trauma, with a normal fundus. Where is the nerve injured, and what drives the damage?
- References (1/2)
- References (2/2)
- Neuro-Ophthalmology